By Jorge Ortiz, Jason André
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85; pp. 1099–1104. Anane, S. & Attouchi, H. (2010). Microsporidiosis: epidemiology, clinical data and therapy. , 34; pp. 450-464. ; Vandeputte, M. (1997). Activities of various compounds against murine and primate polyomaviruses. , 41; pp. 587-593. ; & Gbaji, A. (2003). Malaria prophylaxis in post renal transplant recipients in the tropics: is it necessary? , 49; pp. 63-66. ; Campise, MR, et al. (2005). Natural history of hepatitis B and C in renal allograft recipients. , 79; pp. 1132–1136. ; Pescovitz, MD, et al.
Tropicalis, and C. parapsilosis. Speciation is clinically useful because nonalbicans Candida species vary in in-vitro susceptibility to amphotericin B and azoles (10). , oral thrush, esophageal infection, cutaneous infection at intertriginous sites, candidal vaginitis) is most common in diabetics, with highdose steroid therapy, and during broad-spectrum antibacterial therapy. These infections are usually treatable through correction of the underlying metabolic abnormality and topical therapy with clotrimazole or nystatin without associated risks that may be present for systemically absorbed antifungal agents.
Hepatitis C virus infection and de novo glomerular lesions in renal allografts. , 1; pp. 171-178. ; Gil-Vernet, S. & Grinyó, JM. (2003). Pretransplant interferon prevents hepatitis C virus-associated glomerulonephritis in renal allografts by HCV-RNA clearance. , 3; pp. 357-360. ; Eveleigh, PC. & Van Egmond, JG. (1980). Schistosoma mansoni: characterization of two circulating polysaccharide antigens and the immunological response to these antigens in mouse, hamster, and human infections. , 50; pp.
After the Kidney Transplant - The Patients and Their Allograft by Jorge Ortiz, Jason André